粮油食品科技

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t9,t11-CLA抗大鼠氧化应激损伤的机理研究
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Mechanisms of t9,t11-CLA against oxidative stress injury in rats
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    摘要:

    以大鼠为研究对象,以大鼠血清和肝组织中超氧化物歧化酶(SOD)、谷胱甘肽过氧化物酶(GSH-Px)活性和丙二醛(MDA)含量为指标,研究t9,t11-CLA的体内抗氧化作用效果。结果表明,喂养4周后, t9,t11-CLA添加组中的中剂量组、高剂量组大鼠血清和肝组织中的SOD和GSH-Px活性明显高于正常组(P<0.05),MDA含量的变化则与之相反,说明t9,t11-CLA对大鼠氧化应激损伤有较好的的保护作用,且产生这种保护作用的原因一方面是激活了机体抗氧化酶类,另一方面与t9,t11-CLA的抗氧化能力有关。

    Abstract:

    The antioxidant effect of t9, t11-CLA was investigated based on the indicators of the activities of superoxide dismutase (SOD), glutathione peroxidase (GSH-Px) and the content of malondialdehyde (MDA) in rats serum and liver tissue. The results showed that after feeding for 4 weeks, the activities of SOD and GSH-Px in the serum and liver tissues of middle and high dose group were distinctly higher than that of control group(P<0.05), while the change of the content of MDA was opposite, which indicated that t9, t11-CLA had good protective effect on oxidative stress injury in rats and this protective effect came from their own antioxidant capacity as well as the activated antioxidant enzymes of the body.

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钟先锋,黄建锋,张继如,张勇,黄桂东,华东. t9,t11-CLA抗大鼠氧化应激损伤的机理研究[J].粮油食品科技,2015,23(3):50-52.

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  • 在线发布日期: 2015-09-06
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